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Informationen zum Autor Ved Chauhan, Abha Chauhan, Ted Brown Klappentext This volume collects work from researchers working at the frontier of neuroscience and medicine who report on evidence indicating links between autism and a number of oxidative stress-related abnormalities such as neuroinflammation! mitochondrial dysfunction! and signal transduction. Zusammenfassung The CDC issued an alert indicating that autism may impact one out of every 150 children. This book collects work from researchers who report on evidence indicating links between autism and a number of oxidative stress-related abnormalities such as neuroinflammation, membrane and metabolism abnormalities, and signal transduction. Inhaltsverzeichnis Type, Topography, and Sequelae of Neuropathological Changes Shaping Clinical Phenotype of Autism. Evidence for Oxidative Damage in the Autistic Brain. Oxidative Stress and Neurotrophin Signaling in Autism. Genetics of Autism. Phenotypic Expression of Autism, Maternal Depression, and the Monoamine Oxidase-A Gene. Paraoxonase 1 Status, Environmental Exposures, and Oxidative Stress in Autism Spectrum Disorders. The RedoxMethylation Hypothesis of Autism: A Molecular Mechanism for Heavy Metal-Induced Neurotoxicity. Autism and Oxidative Stress: Evidence from an Animal Model. Neurotoxic Brainstem Impairment as Proposed Threshold Event in Autistic Regression. Abnormalities in Membrane Lipids, Membrane-Associated Proteins, and Signal Transduction in Autism. Mitochondrial Component of Calcium Signaling Abnormality in Autism. Inflammation and Neuroimmunity in the Pathogenesis of Autism: Neural and Immune Network Interactions. Possible Impact of Innate Immunity on Autism. Autism, Gastrointestinal Disturbance, and Immune Dysfunction: What Is the Link. Possible Mechanism Involving Intestinal Oxytocin, Oxidative Stress, and Signaling Pathways in a Subset of Autism with Gut Symptoms. Cytokine Polymorphisms in Autism: Their Role in Immune Alterations. Autism, Teratogenic Alleles, HLA-DR4 , and Immune Function. Autism: The Centrality of Active Pathophysiology and the Shift from Static to Chronic Dynamic Encephalopathy. A Reevaluation of the State of Autism Treatment: The Need for a Biopsychosocial Perspective. ...