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Periodontal diseases are inflammatory diseases of supporting structures of the tooth. It results in the destruction of the supporting structures and most of the destructive processes involved are host derived. The processes leading to destruction and regeneration of the destroyed tissues are of great interest to both researchers and clinicians. The selective susceptibility of subjects for periodontitis has remained an enigma and wide varieties of risk factors have been implicated for the manifestation and progression of periodontitis. Genetic factors have been a new addition to the list of risk factors for periodontal diseases. With the availability of human genome sequence and the knowledge of the complement of the genes, it should be possible to identify the metabolic pathways involved in periodontal destruction and regeneration. Most forms of periodontitis represent a life-long account of interactions between the genome, behaviour, and environment. The current practical utility of genetic knowledge in periodontitis is limited. The information contained within the human genome can potentially lead to a better understanding of the control mechanisms.
About the author
Dr. Arpit Sikri hat sein Studium (BDS) und seine Postgraduiertenausbildung (MDS) im Fach Prothetik abgeschlossen. Während seiner gesamten akademischen Laufbahn wurde er mit Goldmedaillen ausgezeichnet und war der beste Absolvent der Universität. Er hat verschiedene nationale Veröffentlichungen vorzuweisen. Er hielt Vorträge und Posterpräsentationen auf verschiedenen Konferenzen.
